Getting a New mouse. Posted: Thu Nov 21, am. My other one is messed up and to be honest,I am not into laser mouses anyways.
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For patients in the NR group, the distribution of lymphocyte subsets was similar in the peripheral blood of both the patient and the corresponding humanized mice. Thus, ICL cellular pathogenesis might be classified by humanized mouse models into 3 distinct subtypes: a T cell extrinsic, b T cell intrinsic affecting proliferation, and c T cell intrinsic affecting survival. Humanized mouse models of ICL help to delineate etiology and ultimately to guide development of individualized therapeutic strategies. Most ICL cases are detected in adulthood after patients suffer opportunistic, recurrent, or long-lasting infections mostly fungal or mycobacterial; ref. Twenty-five years after the CDC defined ICL, its etiology remains elusive, and there is currently no efficacious treatment, other than the use of antibiotics for prophylaxis or treatment of opportunistic infections At the cellular level, multiple causes have been suggested to be responsible for ICL, such as a defect of hematopoietic stem cells HSCs to create lymphoid precursors 16 , low thymic production 17 , defective homeostatic- or antigen-driven proliferation 15 or survival, 18 and differential migration to tissues
Sets of genes expressed in the same tissue are believed to be under the regulation of a similar set of transcription factors, and can thus be assumed to contain similar structural patterns in their regulatory regions. Here we present a study of the structural patterns in promoters of genes expressed specifically in 26 human and 34 mouse tissues. For each tissue we constructed promoter structure models, taking into account presences of motifs, their positioning to the transcription start site, and pairwise positioning of motifs. Models with high performance include those for liver, skeletal muscle, kidney and tongue. Many of the important structural patterns in these models involve transcription factors of known importance in the tissues in question and structural patterns tend to be conserved between human and mouse. In addition to that, promoter models for related tissues tend to have high inter-tissue performance, indicating that their promoters share common structural patterns. Together, these results illustrate the validity of our models, but also indicate that the promoter structures for some tissues are easier to model than those of others. The activity of genes and their products is regulated on many levels.
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